Lli intervention research paper - syneo-renovation.fr.
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Genetic Separation of the Human Lacritin Gene (“LACRT”) and Triple A (Allgrove) Syndrome on 12Q13. Authors; Authors and affiliations; Rajesh Kumar; Angela Huebner; Gordon W. Laurie; Chapter. 9 Citations; 155 Downloads; Part of the Advances in Experimental Medicine and Biology book series (AEMB, volume 506) Abstract. Molecular mechanisms underlying the pathogenic decline of secretory.
Lacritin is a mitogen of human salivary gland cells as well as a stimulator of human corneal epithelial cells. It is expected to be an important factor in maintaining the surrounding ocular surface. The monkey would be a relevant animal model in which to study the role of lacritin in ophthalmic physiology and pathology. However, to our knowledge, no cDNA cloning or functional analysis of.
Lacritin is a common glycoprotein of the human tear film.It is mainly produced by the lacrimal gland.Some lacritin also is produced by the meibomian gland, and also by epithelial cells of the conjunctiva and cornea.The lacritin gene LACRT is one of the most highly transcriptionally regulated genes in the human eye. Functional studies suggest a role in epithelial renewal of some nongermative.
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Lacritin, a glycoprotein secreted from lacrimal acinar cells, may function as an autocrine factor to stimulate tear protein secretion. The purpose of the present experiment was to investigate lacritin-induced protein secretion in normal and cytokine-pretreated (inflammation model) monkey acinar cells. Acinar cells from monkey lacrimal glands were cultured with or without tumor necrosis factor.
Lacritin levels were then analyzed by ELISA and Western blotting. Results: Anti-Pep Lac N-Term bound lacritin, but not truncation mutant N-65 that lacks the N-terminal antigenic site. Tear.